Like all essential trace minerals selenium serves myriad functions. Of particular note here:
Food sources of selenium include pasture-raised eggs, mushrooms, shellfish, meat (including organ meats), as well as seeds.
Although most Americans get the recommended daily allowance (RDA) of selenium, the late Dr. Gerhard Schrauzer, a world renowned expert and researcher on selenium, said in a 2010 interview: “…for effective cancer risk reduction, selenium supplementation at reasonable levels in adults for example, 100-200 mcg per day, should begin as early as possible and be maintained over the entire lifespan.”
One drop of Lipid-bound Selenium provides 210 mcg. Three drops of Lipid-bound Selenium proves 420 mcg. 400 mcg per day is considered safe upper limit of supplemental selenium.
Lipid-bound Selenium (LbSe) is different from other selenium supplements. While selenium in selenomethionine for example is incorporated in a protein, it is incorporated in a fatty acid in LbSe. Selenoproteins are enzymes, LbSe is not. Selenoproteins can accumulate in all cells, healthy and otherwise, which explains their toxicity. Since LbSe is delivered by free fatty acids almost exclusively to abnormal cells, and any excess is excreted in urine and feces, it has no known toxicity.
Some cancerous cells are anoxybiotic, anaerobic, while others are dysoxybiotic, catabolic. LbSe is toxic for anaerobic cancerous cells but can promote catabolic cells. Follow simultaneous serum and RBC potassium measurements
to determine if higher doses than recommended here should be used in individuals with cancer. Higher doses are recommended if those recommended here fail to solve an anabolic, or anoxybiotic off-balance. Since RBC potassium test results can take days, look for increases in serum potassium in the interim to confirm if the wanted shift is occurring.
While there are no studies of toxicity for long-term use of LbSe, high doses have no known toxicity when used acutely for withdrawal from opioids. Lipid-bound Sulfur (LbS), thiosulfate (MGS) and fatty alcohols, Flame Quell, are often used concurrently to manage symptoms of withdrawal.
Opioids deprive cells of oxygen. Pathological fatty acids are the body’s attempt, albeit ineffectual and therefore exaggerated, to restore oxygen to cells. The symptoms from sudden cessation of an intoxicant are caused by the persistence of pathological fatty acids. LbSe, LbS and thiosulfate oxidize pathological fatty acids while fatty alcohols neutralize them by polar bonding and stearic coupling.
Use therapeutic lipids as necessary for symptom control during withdrawal and wean as symptoms decrease. Maximum suggested dose schedule for opioid withdrawal:
Every four hours rotate through the following:
Hour 1: one dropper lipid-bound Selenium
Hour 2: two droppers MGS
Hour 3: one dropper lipid-bound Sulfur
Hour 4: two droppers Flame Quell
NOTE: droppers of MGS and Flame Quell can be taken in water while droppers of Selenium and Sulfur can be placed directly in mouth or taken on bites of food. Alternatively, a dropper of all agents here will fit in the large end of a double 00 capsule; empty the dropper into the large end, cover with small end and take/administer immediately with beverage or food.<< BACK
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